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Pantoprazole is used in the treatment of barrett's esophagus; gerd; erosive esophagitis; dumping syndrome; helicobacter pylori infection (and ), and belongs to the drug class proton pump inhibitors.

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There is no evidence that acid suppression therapy with PPIs causes regression of Barrett's esophagus or prevents progression to adenocarcinoma of the esophagus.

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The safety and effectiveness of pantoprazole sodium delayed-release tablets for short-term treatment (up to eight weeks) of erosive esophagitis (EE) associated with GERD have been established in pediatric patients 1 year through 16 years of age.

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Given known pharmacokinetics regarding rabeprazole activation, we postulated that a single dose of rabeprazole, given prior to a standard Western meal, would yield greater acid output inhibition and a greater pH on the first day of assessment when compared to a single dose of pantoprazole.

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The most common immune response to a drug is due to the expansion of T cells, a type of white blood cell that recognize the drug as foreign.

In comparisons with other active treatments, pantoprazole has demonstrated high remission rates.

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As in Japan, they are more frequently used when patients are in severe pain from a disease like cancer.

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Enzyme-based interactions are unlikely given the metabolic pathway of raltegravir; however, raltegravir has greatly increased solubility at increased pH and is a substrate to P-glycoprotein, which is at least modestly inhibited by omeprazole, both effects probably being synergistic.

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No side effects that I know of, however my father (who also takes protonix) had to discontinue his regimen for a week because of a test he had to have done said his acid reflux came back about 2 days after he stopped and it was 10x worse than it was before treatment.

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